Doctors will insist upon western medications. These include drugs like Reglan, Domperidone (can't get the United States, must order outside of country), Erithromycin (an antibiotic that causes peristalsis), etc.
However, there are drugs that aren't mentioned that may provide relief.
For instance. A treatment I occasionally will take is Aloe Vera Juice. This aloe vera juice is made from aloe vera gel. This is important because there have been health risks from aloe vera juice containing aloin. Aloe Vera is a natural substance that has healing properties. Most people use it for skin treatments. However, not many people are aware that aloe vera also has has healing properties within the body. Aloe vera juice is one of those juices that can be safely drank 8 oz. at a time, once a day. This is all that is needed for maximum benefit. This will help with irritations and pain caused from a stomach infection. Word of caution: I find aloe vera juice to taste NASTY. However, I have found ways to make it taste better, including mixing it with apple juice (equal parts), and adding a Crystal Light Fruit Punch packet to it. It even has a little effervescence. :)
Another thing I take for it is ginger. However, for ginger to have any type of gastric motility improvement, you have to take about 1200 mg when you eat. The pills I have found here are anywhere from 500 mg to 550 mg. So, I take two of those with ginger ale. This has seemed to help me feel hungry sooner and has helped me pass food a little faster. Ginger is also supposed to be an antiemetic, but for me at least, I don't get that benefit from it.
Another thing I will occasionally take is Iberogast. This is a herbal concoction that you mix with water. About 20 drops taken 2-3 times a day are supposed to help with gastric motility. This stuff even says on the bottle that it is for improving gastric motility. Iberogast has no reported side effects (although if you have problems with alcohol including alcoholism, then don't take). However, for people who can handle a little alcohol and can keep water down, Iberogast has helped a lot of people, including myself.
Betaine HCL with Pepsin is something else I take. This was addressed in Theory #1. I have found that Betaine HCL helps make foods that are normally difficult to digest easier to digest for me. Others may have the same benefits, others may not. However, with this, I would make sure you have low stomach acid before you try it.
I also take Benadryl. Yes, this does classify as a "western medication." However, its usage was NOT prescribed by a doctor. Benadryl's active ingredient is diphenhydramine. Diphenhydramine is a drug found in Sominex and Unisom as well. However, a little known fact. Dramamine is a well-known anti-emetic medication for people with motion sickness. Dramamine's active ingredient is listed as dimenhydrinate. Upon further research, you would find that dimenhydrinate is a combination of two salts: diphenhydramine and 8-chlorotheophylline. 8-chlorotheophylline is in simplest terms, a stimulant similiar to caffeine. So, believe it or not, Benadryl is a safe drug that can be taken to manage nausea. And taking two of those when I am nauseated, does seem to work. Side effect... I get a little drowsy. So I also take meclozine (Draminine II) as an alternative when I am at work.
I have also heard good things of acupuncture as well. I am in the process of doing more research into "at-home" pressure point stimulation of points to improve gastric motility (since I am nowhere near a acupuncturist).
Doctors will mainly focus on western medications because that's what they know. However, people have survived and dealt with a lot of these problems long before western medication. As such, we should be able to rely on older methods to combat these issues as well. Let me know how eastern intervention has helped you. :)
As always, thank you for reading. :)
Thursday, May 30, 2013
Theory #3 - Exercise helps
Gastroparesis is caused by damage to the vagus nerve. The vagus nerve is responsible for proper peristalsis (wave-like contractions of the stomach muscle responsible for moving food down the stomach). If the vagus nerve is damaged, then the peristalsis will be weaker than required for proper food break-down and digestion. Now, I have addressed low stomach acid in an earlier theory (theory 1). However, with gastroparesis, proper stomach movement is also important in breaking down food and moving it to the next level of digestion.
For this theory, we will look at multiple sclerosis as a point here. A lot of people diagnosed with multiple sclerosis are required to start up an intense exercise regimen to regain or maintain muscle control. Montel Williams was a classic example of this when he went on Oprah many years ago and described his intense workouts. His workouts allowed him the ability to walk and function in society, despite the pain. If he hasn't done the exercise, he wouldn't have maintained the ability to walk like he has.
So, another possible theory is that the same can be applied for gastroparesis.
The stomach is more than just an organ. It is more than just a pouch for food storage. It has muscles. Smooth muscles, nonetheless, but muscles. People can exercise the heart. People can exercise the brain. Why couldn't people exercise the stomach?
It has moveable components. It has contractions. So, what if we could make those contractions stronger? Naturally. I'm not talking using medications to stimulate stomach contractions (as many of those medications contain harmful side effects). I'm talking healthier approaches.
Now, a lot of people with gastroparesis have a lot of weight loss issues. They are too skinny. Or vice versa, they are overweight. Some of them are of healthy weight. Gastroparesis effects everyone differently. In the case of people who are too skinny, the challenge is: coming up with exercises that focus only on the stomach, and don't burn too many calories. Face it, if you are too skinny, you want to hold on to every calorie you can get.
So, someone had noted that horseback riding is an excellent exercise in building core muscles including stomach muscles. Increasing stomach muscles can increase stomach motility. However, what if you can't get access to a horse? What exercises can you do?
Well, for one. There is a belly roll that can be done. This involves distending the stomach out as far as possible, and then rolling it back in as tight as you can in an downward movement. And then do it in reverse. Keep doing this until the stomach gets tired. This is an exercise that can stimulate gastric motility.
Other exercises that can be done, involve doing crunches on the floor, or with an exercise ball. Walking is also another good exercise that improves gastric motility.
What exercises have you tried? Have any worked for you? Comment below.
Thank you for reading. :)
Dr. Bernstein's Diabetes Solutions - Exercises that Facilitate Stomach Emptying
For this theory, we will look at multiple sclerosis as a point here. A lot of people diagnosed with multiple sclerosis are required to start up an intense exercise regimen to regain or maintain muscle control. Montel Williams was a classic example of this when he went on Oprah many years ago and described his intense workouts. His workouts allowed him the ability to walk and function in society, despite the pain. If he hasn't done the exercise, he wouldn't have maintained the ability to walk like he has.
So, another possible theory is that the same can be applied for gastroparesis.
The stomach is more than just an organ. It is more than just a pouch for food storage. It has muscles. Smooth muscles, nonetheless, but muscles. People can exercise the heart. People can exercise the brain. Why couldn't people exercise the stomach?
It has moveable components. It has contractions. So, what if we could make those contractions stronger? Naturally. I'm not talking using medications to stimulate stomach contractions (as many of those medications contain harmful side effects). I'm talking healthier approaches.
Now, a lot of people with gastroparesis have a lot of weight loss issues. They are too skinny. Or vice versa, they are overweight. Some of them are of healthy weight. Gastroparesis effects everyone differently. In the case of people who are too skinny, the challenge is: coming up with exercises that focus only on the stomach, and don't burn too many calories. Face it, if you are too skinny, you want to hold on to every calorie you can get.
So, someone had noted that horseback riding is an excellent exercise in building core muscles including stomach muscles. Increasing stomach muscles can increase stomach motility. However, what if you can't get access to a horse? What exercises can you do?
Well, for one. There is a belly roll that can be done. This involves distending the stomach out as far as possible, and then rolling it back in as tight as you can in an downward movement. And then do it in reverse. Keep doing this until the stomach gets tired. This is an exercise that can stimulate gastric motility.
Other exercises that can be done, involve doing crunches on the floor, or with an exercise ball. Walking is also another good exercise that improves gastric motility.
What exercises have you tried? Have any worked for you? Comment below.
Thank you for reading. :)
Dr. Bernstein's Diabetes Solutions - Exercises that Facilitate Stomach Emptying
Theory #2 - Gastroparesis isn't primary.
Another thing that has boggled my mind with gastroparesis is this... Gastroparesis has been diagnosed as idiopathic in 30% - 50% of cases (depending on your resource). Now, let us analyze what idiopathic means.
Idiopathic = no known cause.
Now, the majority of cases of gastroparesis are diabetes related. This is one I can understand. Diabetes causes nerve damage, which in turn causes gastroparesis (when the vagus nerve gets damaged). However, there are A LOT of causes of gastroparesis.
Which means, A LOT of cases of gastroparesis have a secondary cause, that just hasn't been discovered yet.
For instance, when I was diagnosed with idiopathic gastroparesis, I had only two tests. An endoscopy, and a gastric emptying scintigraphy. And then that was it. Diabetes had been ruled out because my blood sugars were normal during the blood test.
However. Gastroparesis has several known causes (and I'm sure a lot more unknown causes). These include:
But I feel that gastroparesis doesn't just... happen. I feel gastroparesis HAS to have been caused by something. Something caused damage to the vagus nerve. Something caused this to happen. I feel it can not just be primary. It must be a secondary problem.
Which leads us to the importance of finding the primary cause. This primary cause governs EVERYTHING. Treatment options (while primarily thought to be the same for all cases of gastroparesis), are actually very different depending on the cause. For instance, gastroparesis caused by overuse of medications (including medications containing acetaminophen) can actually go away by ceasing use of said medications. Gastroparesis caused by a bad stomach bug, have in some reports gone away after about a year or more. Gastroparesis caused by a chronic health condition, however, doesn't. For instance, if you had multiple sclerosis and it caused your gastroparesis, then when your MS triggers, your gastroparesis triggers (and will of course get worse as it does).
So, finding the cause of gastroparesis, also determines its eventual prognosis. If we could find out what caused it, and how to treat the underlying cause, we may be able to treat the gastroparesis as well.
Interesting theory.
So, what caused yours?
I believe mine was caused by a stomach bug I developed as a result of low stomach acid. So, I am hoping to heal from this as a result.
Hoping to hear from you soon. :)
Idiopathic = no known cause.
Now, the majority of cases of gastroparesis are diabetes related. This is one I can understand. Diabetes causes nerve damage, which in turn causes gastroparesis (when the vagus nerve gets damaged). However, there are A LOT of causes of gastroparesis.
Which means, A LOT of cases of gastroparesis have a secondary cause, that just hasn't been discovered yet.
For instance, when I was diagnosed with idiopathic gastroparesis, I had only two tests. An endoscopy, and a gastric emptying scintigraphy. And then that was it. Diabetes had been ruled out because my blood sugars were normal during the blood test.
However. Gastroparesis has several known causes (and I'm sure a lot more unknown causes). These include:
- Multiple Sclerosis
- Hypothyroidism
- Surgery
- Acid Reflux
- Parkinson's Disease
- prior stomach bug
- Medications
- Scleroderma
- Amyloidosis
- list goes on...
But I feel that gastroparesis doesn't just... happen. I feel gastroparesis HAS to have been caused by something. Something caused damage to the vagus nerve. Something caused this to happen. I feel it can not just be primary. It must be a secondary problem.
Which leads us to the importance of finding the primary cause. This primary cause governs EVERYTHING. Treatment options (while primarily thought to be the same for all cases of gastroparesis), are actually very different depending on the cause. For instance, gastroparesis caused by overuse of medications (including medications containing acetaminophen) can actually go away by ceasing use of said medications. Gastroparesis caused by a bad stomach bug, have in some reports gone away after about a year or more. Gastroparesis caused by a chronic health condition, however, doesn't. For instance, if you had multiple sclerosis and it caused your gastroparesis, then when your MS triggers, your gastroparesis triggers (and will of course get worse as it does).
So, finding the cause of gastroparesis, also determines its eventual prognosis. If we could find out what caused it, and how to treat the underlying cause, we may be able to treat the gastroparesis as well.
Interesting theory.
So, what caused yours?
I believe mine was caused by a stomach bug I developed as a result of low stomach acid. So, I am hoping to heal from this as a result.
Hoping to hear from you soon. :)
Wednesday, May 29, 2013
Theory #1 - Low stomach acid in regards to gastroparesis
This is a thought that has boggled my mind for quite a while. This is a KEY in maintaining my condition. I take Betaine HCL with Pepsin.
No gastroenterologist has ever diagnosed me with low stomach acid. I had spent a long amount of time believing that acid reflux = too much stomach acid. I had no idea that LOW stomach acid could also cause acid reflux.
With low stomach acid, the stomach churns causing that acid to splash more. Low acid = more splash. More splash = burning in the esophagus. So, I treated my acid reflux with Prilosec (omeprazole). However, I started to get sick a lot more. In the winter of 2011, I started vomiting (something I hadn't done since the year 2000). I ended up throwing up about 5-6 times from Nov to Apr (2011-2012). And then it stopped for a while (I also eased up on the Prilosec). And then I went back on the Prilosec in the fall of 2012 due to bad acid reflux. I was also on a higher dosage (40 mg instead of the 20 mg I used to take). In December 2012, I got really sick. I mean throwing up for about an hour every 5 minutes, and then a 24 hour spell of diarrhea to follow. My gastroparesis story technically started here...
Now, no one else around me had a stomach bug of any kind. I didn't catch it from any one around me. In fact, this was before the big norovirus season started. So, this led me to do research on low stomach acid. I'm guessing that there was a bug in the food I ate. Possibly a botulism-related virus. Now, botulism is especially hard on infants (due to low stomach acid). But it can also be bad for an adult if they had been taking acid reflux medication.
This led me to the conclusion, I must have had low stomach acid. I even did the "burp" test. [read about that test here.] After 5 minutes, I did not belch at all and stopped the test.
I then started taking Betaine HCL, and my acid reflux is pretty much under control. So, that's good. Now, how does this relate to gastroparesis? This is the basis for the theory.
Stomach acid is a natural defense mechanism. It kills bacteria that may be in the stomach. That's right. It KILLS bacteria. So, my theory is thus: Increasing stomach acid helps prevent bacterial overgrowth.
Low stomach acid turns the stomach into a perfect breeding ground for bacteria. Food that sits in the stomach for long periods of time develop bacteria. On top of that, bacteria also enters your stomach as reflux from the small intestine. On top of that, bacteria also enters your stomach from the saliva you swallow. So, the stomach constantly gets bombarded with bacteria. So, all of this bacteria causes food to rot. The bi-product of bacteria is... gas. This results in bloating. Why do you think that rotting road kill bloats?
On top of that stomach acid helps break down food. Gastroenterologists may tell you that food doesn't move due to irregular peristalsis (irregular stomach contractions), but they will never mention that low stomach acid can be a contributing factor. Basically, if your stomach acid is low, your body will attempt to pass undigested or larger chucks of food. This makes digestion much harder. So, increasing your stomach acid levels would theoretically make some of the harder foods to digest, easier to digest.
So, now, with every meal (even if it IS on the gastroparesis diet), I will always take a betaine HCL pill with it. I get the kind that contains Pepsin because Pepsin helps the stomach produce more acid. And I must say, I haven't been nauseated NEARLY as often as I used to be.
So, some of your gastroparesis sufferers out there... It might be recommended to get tested for low stomach acid. If your stomach acids are low, you might benefit from adding betaine HCL to your diet.
:) Hoping to hear from you. :) Let me know if this is something you have tried and whether it works for you.
- Nathan S
No gastroenterologist has ever diagnosed me with low stomach acid. I had spent a long amount of time believing that acid reflux = too much stomach acid. I had no idea that LOW stomach acid could also cause acid reflux.
With low stomach acid, the stomach churns causing that acid to splash more. Low acid = more splash. More splash = burning in the esophagus. So, I treated my acid reflux with Prilosec (omeprazole). However, I started to get sick a lot more. In the winter of 2011, I started vomiting (something I hadn't done since the year 2000). I ended up throwing up about 5-6 times from Nov to Apr (2011-2012). And then it stopped for a while (I also eased up on the Prilosec). And then I went back on the Prilosec in the fall of 2012 due to bad acid reflux. I was also on a higher dosage (40 mg instead of the 20 mg I used to take). In December 2012, I got really sick. I mean throwing up for about an hour every 5 minutes, and then a 24 hour spell of diarrhea to follow. My gastroparesis story technically started here...
Now, no one else around me had a stomach bug of any kind. I didn't catch it from any one around me. In fact, this was before the big norovirus season started. So, this led me to do research on low stomach acid. I'm guessing that there was a bug in the food I ate. Possibly a botulism-related virus. Now, botulism is especially hard on infants (due to low stomach acid). But it can also be bad for an adult if they had been taking acid reflux medication.
This led me to the conclusion, I must have had low stomach acid. I even did the "burp" test. [read about that test here.] After 5 minutes, I did not belch at all and stopped the test.
I then started taking Betaine HCL, and my acid reflux is pretty much under control. So, that's good. Now, how does this relate to gastroparesis? This is the basis for the theory.
Stomach acid is a natural defense mechanism. It kills bacteria that may be in the stomach. That's right. It KILLS bacteria. So, my theory is thus: Increasing stomach acid helps prevent bacterial overgrowth.
Low stomach acid turns the stomach into a perfect breeding ground for bacteria. Food that sits in the stomach for long periods of time develop bacteria. On top of that, bacteria also enters your stomach as reflux from the small intestine. On top of that, bacteria also enters your stomach from the saliva you swallow. So, the stomach constantly gets bombarded with bacteria. So, all of this bacteria causes food to rot. The bi-product of bacteria is... gas. This results in bloating. Why do you think that rotting road kill bloats?
On top of that stomach acid helps break down food. Gastroenterologists may tell you that food doesn't move due to irregular peristalsis (irregular stomach contractions), but they will never mention that low stomach acid can be a contributing factor. Basically, if your stomach acid is low, your body will attempt to pass undigested or larger chucks of food. This makes digestion much harder. So, increasing your stomach acid levels would theoretically make some of the harder foods to digest, easier to digest.
So, now, with every meal (even if it IS on the gastroparesis diet), I will always take a betaine HCL pill with it. I get the kind that contains Pepsin because Pepsin helps the stomach produce more acid. And I must say, I haven't been nauseated NEARLY as often as I used to be.
So, some of your gastroparesis sufferers out there... It might be recommended to get tested for low stomach acid. If your stomach acids are low, you might benefit from adding betaine HCL to your diet.
:) Hoping to hear from you. :) Let me know if this is something you have tried and whether it works for you.
- Nathan S
Welcome to my Gastroparesis Theories Blog...
Welcome to this blog. First of all, allow me to introduce myself.
I am Nathan Swanson. I am 30 years old as of the date of this posting. I was diagnosed with gastroparesis in March of 2013 (so, not that long ago). Before my diagnosis, I would have week long spells of nausea. I would feel fine one week, and then feel like throwing up for about a week, and then start feeling better. During each of these spells, I would lose 15 lbs. This had been happening on and off since Dec 8, 2012. The year before that, I had gotten sick to my stomach about 5-6 times. And before that, I hadn't been sick to my stomach in over 10 years (but I had a lot of acid reflux issues).
Around the times I was getting sick, I was taking omeprazole (Prilosec) for acid reflux issues. This comes into play eventually...
December 15, 2012, started out as a normal day for me. Felt fine most of the day. That night, our company had a Christmas party. I ate a decent meal with everyone. About 45 minutes to an hour after the meal, I started feeling very nauseated. I ended up leaving the party early and got across the street and then vomited. I managed to walk home, and then proceeded to spend an hour on and off vomiting about every 5 minutes. I figured that I must have had contracted some serious stomach bug.
Ever since then, I would feel fine, and then get the week of nausea. This leads me to believe that my gastroparesis was a result of this stomach bug that I had gotten.
I didn't get diagnosed with it until March when I finally decided to let my health insurance start working for me.
A lot of the people I had met with this disorder, had doctors claim that their illness was "in their heads." I was fortunate to get a referral to the gastroenterology clinic in Sioux Falls. After two tests, an esophagogastroduodenoscopy (upper gastric endoscopy), and a gastric emptying scintigraphy, I had my diagnosis, and a lot of confusion.
So, naturally, being of an insatiable sense of curiosity and my obsessive need to research and study EVERYTHING medical (I'm a hypochondriac)... I did a lot of research. I pretty much know a lot about this disorder. I have gone over medical studies and journals. I have done a lot of eastern treatments. I have joined a Facebook group of people who also have this disorder. And I am trying to stay positive.
I am here to report that I haven't had many bad flare-ups since I started trying things. I can eat most foods without issues (although, there are some foods that I have discovered will incapacitate me with nausea for a day or two). I find it hard to actually keep to the gastroparesis diet that was provided with me after my diagnosis. But I figure, as long as what I'm doing works... why should I? I am keep up with my nutritional needs. I can even eat nuts (a big no-no as nuts don't digest), as long as I do so in moderation. I can't eat hoards and hoards of nuts like I used to. Oh well.
Now, anyone looking at me, would not be able to tell I have this problem. I am overweight. I used to tell myself when I was nauseated for a week at a time, "Oh look, I lost 15 lbs. I could definitely use the weight loss, I just don't want to lose it THIS way." Being that I'm a hypochondriac as well, I have a tendency to overreact, and my fear of the rapid weight loss was... If I lose this weight too fast, I'll develop ketoacidosis (which can develop in anorexia and bulimic people as well as diabetics).
The medical reports I have read all ended in disappointment. The studies were cancelled prematurely in my opinion. However, they did provide some insight. And they allowed me to formulate theories. My mind always is coming up with theories and thoughts that I think are genius. I decided I had enough of just thinking of this stuff... I'm going to start posting it. Either others will share in my thoughts, or others will criticize. I am hoping that readers of this will share their input. Remember, I am not a doctor. My theories will sometimes border on the medical side, but being that there isn't a lot known about this disorder, a lot of the gastroparesis sufferers out there probably know just as much (if not more) than the doctors themselves. The doctors can test for it, but the sufferers truly know how it feels and how it works from a more personal perspective. But being that I'm not a doctor, if one of my theories just doesn't seem to agree with what you were told, then don't yell at me about it. Gastroparesis affects everyone differently. What works for one person will not work for another. With that in mind, good luck and I hope you find this blog helpful.
Also, if you have any theories of your own you would like to share, PLEASE share them with me. I would be glad to include them in this blog. Theories should be shared. Theories get tested... And that's how we define fact. I am looking to band together as a community, sharing ideas and remedies to minimize flare-ups and to improve overall quality of life. Thank you for reading!!! :)
I am Nathan Swanson. I am 30 years old as of the date of this posting. I was diagnosed with gastroparesis in March of 2013 (so, not that long ago). Before my diagnosis, I would have week long spells of nausea. I would feel fine one week, and then feel like throwing up for about a week, and then start feeling better. During each of these spells, I would lose 15 lbs. This had been happening on and off since Dec 8, 2012. The year before that, I had gotten sick to my stomach about 5-6 times. And before that, I hadn't been sick to my stomach in over 10 years (but I had a lot of acid reflux issues).
Around the times I was getting sick, I was taking omeprazole (Prilosec) for acid reflux issues. This comes into play eventually...
December 15, 2012, started out as a normal day for me. Felt fine most of the day. That night, our company had a Christmas party. I ate a decent meal with everyone. About 45 minutes to an hour after the meal, I started feeling very nauseated. I ended up leaving the party early and got across the street and then vomited. I managed to walk home, and then proceeded to spend an hour on and off vomiting about every 5 minutes. I figured that I must have had contracted some serious stomach bug.
Ever since then, I would feel fine, and then get the week of nausea. This leads me to believe that my gastroparesis was a result of this stomach bug that I had gotten.
I didn't get diagnosed with it until March when I finally decided to let my health insurance start working for me.
A lot of the people I had met with this disorder, had doctors claim that their illness was "in their heads." I was fortunate to get a referral to the gastroenterology clinic in Sioux Falls. After two tests, an esophagogastroduodenoscopy (upper gastric endoscopy), and a gastric emptying scintigraphy, I had my diagnosis, and a lot of confusion.
So, naturally, being of an insatiable sense of curiosity and my obsessive need to research and study EVERYTHING medical (I'm a hypochondriac)... I did a lot of research. I pretty much know a lot about this disorder. I have gone over medical studies and journals. I have done a lot of eastern treatments. I have joined a Facebook group of people who also have this disorder. And I am trying to stay positive.
I am here to report that I haven't had many bad flare-ups since I started trying things. I can eat most foods without issues (although, there are some foods that I have discovered will incapacitate me with nausea for a day or two). I find it hard to actually keep to the gastroparesis diet that was provided with me after my diagnosis. But I figure, as long as what I'm doing works... why should I? I am keep up with my nutritional needs. I can even eat nuts (a big no-no as nuts don't digest), as long as I do so in moderation. I can't eat hoards and hoards of nuts like I used to. Oh well.
Now, anyone looking at me, would not be able to tell I have this problem. I am overweight. I used to tell myself when I was nauseated for a week at a time, "Oh look, I lost 15 lbs. I could definitely use the weight loss, I just don't want to lose it THIS way." Being that I'm a hypochondriac as well, I have a tendency to overreact, and my fear of the rapid weight loss was... If I lose this weight too fast, I'll develop ketoacidosis (which can develop in anorexia and bulimic people as well as diabetics).
The medical reports I have read all ended in disappointment. The studies were cancelled prematurely in my opinion. However, they did provide some insight. And they allowed me to formulate theories. My mind always is coming up with theories and thoughts that I think are genius. I decided I had enough of just thinking of this stuff... I'm going to start posting it. Either others will share in my thoughts, or others will criticize. I am hoping that readers of this will share their input. Remember, I am not a doctor. My theories will sometimes border on the medical side, but being that there isn't a lot known about this disorder, a lot of the gastroparesis sufferers out there probably know just as much (if not more) than the doctors themselves. The doctors can test for it, but the sufferers truly know how it feels and how it works from a more personal perspective. But being that I'm not a doctor, if one of my theories just doesn't seem to agree with what you were told, then don't yell at me about it. Gastroparesis affects everyone differently. What works for one person will not work for another. With that in mind, good luck and I hope you find this blog helpful.
Also, if you have any theories of your own you would like to share, PLEASE share them with me. I would be glad to include them in this blog. Theories should be shared. Theories get tested... And that's how we define fact. I am looking to band together as a community, sharing ideas and remedies to minimize flare-ups and to improve overall quality of life. Thank you for reading!!! :)
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